Dr Anurag Singh - Kidney Care
About Me
Dr Anurag Singh - Kidney Care
MBBS
Nephrologist
10 Years Experience
Biography
My Clinic
OPD Hours
| Monday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Tuesday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Wednesday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Thursday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Friday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Saturday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM | |
| Sunday | Morning | 10:00 AM | - | 02:00 PM |
| Evening | 06:00 PM | - | 09:00 PM |
Treatments
Specialised care and treatments offered
RENAL TUBULAR ACIDOSIS (RTA)
This metabolic disorder occurs when kidneys fail to properly acidify urine resulting in systemic acidosis impacting multiple organ systems. Understanding its mechanisms helps guide effective treatment plans targeted at restoring acid-base balance.
- Clinical manifestations : Fatigue , muscle weakness , bone pain due to disturbances in electrolyte balance ; growth retardation possible in children .
- Laboratory findings : Blood tests show low bicarbonate ; urine pH remains abnormally high despite metabolic acidosis confirming tubular defects .
- Causes : Can be inherited genetic defects or acquired secondary conditions like autoimmune diseases or certain medications .
- Treatment principles : Alkali therapy such as oral bicarbonate supplements neutralize excess acid ; correcting underlying disorders when identified .
Timely diagnosis combined with consistent management helps prevent complications like bone demineralization improving overall prognosis for individuals living with RTA .
HYPERTENSIVE NEPHROPATHY
This condition results from long-standing uncontrolled hypertension causing gradual damage to renal blood vessels leading to chronic kidney impairment. Effective management is critical for slowing disease progression.
- Development signs : Persistent high blood pressure readings combined with proteinuria or declining kidney filtration rate raises suspicion .
- Diagnostic evaluation : Comprehensive blood pressure monitoring , urinalysis , serum creatinine measurements , imaging studies like renal Doppler ultrasound assess vessel integrity .
- Treatment modalities : Aggressive blood pressure control using ACE inhibitors /ARBs protects kidneys ; lifestyle adjustments including salt restriction , exercise are vital .
- Complication awareness : Without prompt treatment , it may advance towards end-stage renal disease requiring dialysis .
Early diagnosis coupled with meticulous hypertension management offers the best chances for preserving renal health amid hypertensive nephropathy .
UREMIC ENCEPHALOPATHY
This neurological condition arises when kidneys fail to remove toxins efficiently from the bloodstream leading to brain dysfunction. It is a critical complication seen in advanced kidney failure stages.
- Symptoms observed: Confusion, decreased alertness, tremors, seizures can manifest indicating worsening toxin accumulation affecting brain cells.
- Differential diagnosis tools: Blood urea nitrogen (BUN) tests significantly elevated along with creatinine indicate poor renal clearance; neurological exams assist assessment further.
- Treatment focus areas: Prompt dialysis initiation removes accumulated toxins rapidly; supportive care includes controlling seizures if present;
- Preventative measures : Managing chronic kidney disease progression reduces risk of development;
With rapid recognition and appropriate intervention including dialysis , uremic encephalopathy can be reversed , improving neurological outcome .
NEPHROTIC SYNDROME
The nephrotic syndrome is characterized by significant protein loss through urine due to increased permeability of the glomerular membrane. This leads to a distinct cluster of symptoms requiring timely intervention.
- Main clinical features: Swelling especially in legs and around eyes (edema), foamy urine due to excess protein, fatigue caused by low albumin levels.
- How it’s diagnosed: Urinalysis revealing high protein levels; blood tests indicating low serum albumin; kidney biopsy sometimes necessary for precise cause identification.
- Treatment strategies: Typically involve corticosteroids or other immunosuppressants depending on cause; diuretics help control edema; managing cholesterol levels is also important.
- Monitoring importance: Regular follow-ups ensure treatment response assessment and prevention of complications like infections or blood clots.
The prognosis varies by underlying disease but with proper care many patients achieve remission with restored quality of life.
GLOMERULONEPHRITIS
This group of diseases involves inflammation of the tiny filters in the kidneys called glomeruli. Glomerulonephritis impacts the kidney’s filtering ability leading to various complications.
- Presentation and causes: Symptoms include blood in urine (hematuria), swelling around eyes or legs, high blood pressure. It may stem from infections or autoimmune conditions.
- Diagnostic measures: Urinalysis showing proteinuria or hematuria, blood tests for kidney function and immune markers, and sometimes a kidney biopsy confirm diagnosis.
- Treatment options: Depending on cause severity: immunosuppressant drugs, blood pressure control medications, or antibiotics if infection-related.
- Lifestyle modifications: Dietary changes such as reduced salt intake help manage symptoms and reduce kidney stress.
A comprehensive approach involving medication and careful monitoring aims to preserve kidney function long-term in glomerulonephritis patients.
ACUTE KIDNEY INJURY (AKI)
Acute Kidney Injury (AKI) represents a sudden decline in kidney function, often developing over hours to days. This rapid loss of renal function can affect fluid and electrolyte balance critically.
- Symptoms and causes: Patients may experience reduced urine output, swelling, fatigue, or confusion. AKI can result from dehydration, infections, medications, or blockage in urinary flow.
- Diagnosis: Blood tests measuring creatinine and urea levels, along with urine analysis and imaging studies like ultrasound, help identify AKI.
- Treatment approaches: Management focuses on addressing the underlying cause, ensuring proper hydration, avoiding nephrotoxic drugs, and close monitoring of electrolytes.
- Hospital care requirements: In severe cases, dialysis may be necessary temporarily to support kidney function until recovery.
- Prognosis: Early detection and treatment often lead to complete recovery; however, severe or prolonged injury may progress to chronic kidney issues.
Recognizing the signs early and seeking prompt medical care is vital to prevent lasting damage from AKI. Supportive therapies aim to restore kidney function safely.